The American Journal of Human Genetics has a new preprint up that tentatively suggests that there is a parental specific genomic imprinting associated with FOXP2. I’ve put up the PDF as “foxp2.pdf” in the GNXP Forum. In short, it seems that the paternal copy might be overexpressed relative to the maternal copy. Anyone care to offer an explanation for why the male copy (which would presumably be less genetically close to the other male copies in a sib-cohort) might want FOXP2 overexpressed relative to the female copy?
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